Research into the biggest killer in Britain has uncovered a landmark answers to heart disease and its link to a few rogue genes and choices in lifestyle. These 18 genes have been detected, in three studies that have included hundreds of scientists worldwide, to increase the risk of cardiac problems from hardening of arteries to heart attacks.
Preventing heart disease, blamed for over 12% of death worldwide, now can be better treated and even possibly prevented, possibly lowering the over 90,000 deaths annually in the UK, costing the economy over 9 billion pounds and killing a Briton every six minutes.
Researchers are saying that heart disease could be eliminated within 50 years with the discovery of these genes that increasing the number of known genes that can cause heart disease by 50%. What many of the genes do to effect the heart is still unknown but ones that are include; blood pressure, cholesterol and other processes of the heart.
Scientists are pleased knowing that causes of the disease are yet to be found and drugs to fight against the genes can one day be found and the result will be much improvement to health. With the identification of many genes, this has become a landmark result since most of the genes operate using mechanisms unknown to us now and the job now is to understand how they work and develop new drugs to fight them and also identify the people most benefit by their use.
In three studies the 18 genes were discovered by some 300 scientists from all over the world including Britons analyzing over 200,000 people. Narrowing of the arteries that supply oxygen-rich blood to genetic links was focused on by the scientists. The narrowing is caused by fatty deposits that build up raised the percentages of many illnesses ranging from heart attacks, to heart failure angina and irregular heartbeats and drugs that were made to stop vessels from clogging would reduce profoundly the risk of having a heart attack.
It is thought that in under a decade the first new drugs could be available in the market said Oxford University Professor Hugh Watkins, that co-led the studies.